Evans syndrome is an autoimmune disease in which an individual's immune system attacks their own red blood cells and platelets, the syndrome can include immune neutropenia.[1][2] These immune cytopenias may occur simultaneously or sequentially.[1][3]
Its overall phenotype resembles a combination of autoimmune hemolytic anemia and immune thrombocytopenic purpura.[1][4][5]
Autoimmune hemolytic anemia is a condition in which the red blood cells that normally carry oxygen are destroyed by an autoimmune process. Immune thrombocytopenic purpura is a condition in which platelets are destroyed by an autoimmune process. Platelets are a component of blood that contribute to the formation of blood clots in the body to prevent bleeding.
The syndrome was first described in 1951 by R. S. Evans and colleagues.[1]
^ abcd"Evans Syndrome". NORD (National Organization for Rare Disorders). Retrieved 2022-03-15.
^"Evans syndrome". Genetic and Rare Diseases Information Center. Retrieved 2018-04-17.
^Norton A, Roberts I (2006). "Management of Evans syndrome". Br. J. Haematol. 132 (2): 125–37. doi:10.1111/j.1365-2141.2005.05809.x. PMID 16398647. S2CID 7633446.
^Cite error: The named reference pmid31292991 was invoked but never defined (see the help page).
^Michel M; Chanet V; Dechartres A; Morin A-S; Piette J-C; Cirasino L; Emilia G; Zaja F; Ruggeri M; Andrès E; Bierling P; Godeau B; Rodeghiero F (2009). "The spectrum of Evans syndrome in adults: new insight into the disease based on the analysis of 68 cases". Blood. 114 (15): 3167–3172. doi:10.1182/blood-2009-04-215368. PMID 19638626.
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