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Foot drop information


Foot drop
Shown here, the right foot drops due to paralysis of the tibialis anterior muscle, while the left foot demonstrates normal lifting abilities.
SpecialtyNeurology Edit this on Wikidata
A patient recovering from surgery to treat foot drop, with limited plantar and dorsiflexion.

Foot drop is a gait abnormality in which the dropping of the forefoot happens due to weakness, irritation or damage to the deep fibular nerve (deep peroneal), including the sciatic nerve, or paralysis of the muscles in the anterior portion of the lower leg. It is usually a symptom of a greater problem, not a disease in itself. Foot drop is characterized by inability or impaired ability to raise the toes or raise the foot from the ankle (dorsiflexion). Foot drop may be temporary or permanent, depending on the extent of muscle weakness or paralysis and it can occur in one or both feet. In walking, the raised leg is slightly bent at the knee to prevent the foot from dragging along the ground.

Foot drop can be caused by nerve damage alone or by muscle or spinal cord trauma, abnormal anatomy, toxins, or disease. Toxins include organophosphate compounds which have been used as pesticides and as chemical agents in warfare. The poison can lead to further damage to the body such as a neurodegenerative disorder called organophosphorus induced delayed polyneuropathy. This disorder causes loss of function of the motor and sensory neural pathways. In this case, foot drop could be the result of paralysis due to neurological dysfunction. Diseases that can cause foot drop include trauma to the posterolateral neck of fibula, stroke,[1][2][3][4] amyotrophic lateral sclerosis, muscular dystrophy, poliomyelitis, Charcot–Marie–Tooth disease, multiple sclerosis, cerebral palsy, hereditary spastic paraplegia, Guillain–Barré syndrome, Welander distal myopathy, Friedreich's ataxia and chronic compartment syndrome. It may also occur as a result of hip replacement surgery or knee ligament reconstruction surgery.

  1. ^ Chisholm, Amanda E.; Perry, Stephen D.; McIlroy, William E. (2013-11-01). "Correlations between ankle–foot impairments and dropped foot gait deviations among stroke survivors". Clinical Biomechanics. 28 (9): 1049–1054. doi:10.1016/j.clinbiomech.2013.09.007. ISSN 0268-0033. PMID 24094611.
  2. ^ Stewart, John D. (2008-06-01). "Foot drop: where, why and what to do?". Practical Neurology. 8 (3): 158–169. doi:10.1136/jnnp.2008.149393. ISSN 1474-7758. PMID 18502948. S2CID 28002732.
  3. ^ Pittock, S. J.; Moore, A. P.; Hardiman, O.; Ehler, E.; Kovac, M.; Bojakowski, J.; Khawaja, I. al; Brozman, M.; Kaňovský, P.; Skorometz, A.; Slawek, J.; Reichel, G.; Stenner, A.; Timerbaeva, S.; Stelmasiak, Z. (2003). "A Double-Blind Randomised Placebo-Controlled Evaluation of Three Doses of Botulinum Toxin Type A (Dysport®) in the Treatment of Spastic Equinovarus Deformity after Stroke". Cerebrovascular Diseases. 15 (4): 289–300. doi:10.1159/000069495. ISSN 1015-9770. PMID 12686794. S2CID 29635400.
  4. ^ Gao, Ying; Gang, Xiaochao; Yuan, Yue; Yin, Kai; Gong, Xiaoyan (2022-10-07). "Efficacy and safety of acupuncture in the treatment of foot drop in post-stroke: A protocol for systematic review and meta-analysis". Medicine. 101 (40): e30994. doi:10.1097/MD.0000000000030994. PMC 9542557. PMID 36221406.

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