Transfusional hemosiderosis is the accumulation of iron in the body due to frequent blood transfusions. Iron accumulates in the liver and heart, but also endocrine organs. Frequent blood transfusions may be given to many patients, such as those with thalassemia, sickle cell disease, leukemia, aplastic anemia, or myelodysplastic syndrome, among others. It is diagnosed with a blood transferrin test and a liver biopsy. It is treated with venipuncture, erythrocytapheresis, and iron chelation therapy.
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hemosiderosis can cause cardiac arrhythmia and cardiomyopathy. Transfusional hemosiderosis is a potential side effect of frequent blood transfusions....
Hemosiderosis is a form of iron overload disorder resulting in the accumulation of hemosiderin. Types include: Transfusionhemosiderosis Idiopathic pulmonary...
or hemosiderosis that are occasionally used include: Hemosiderosis is hemochromatosis caused by excessive blood transfusions, that is, hemosiderosis is...
by chemotherapy. Physicians hoped the sterile room and frequent blood transfusions would sustain him until he recovered spontaneously, or an effective treatment...
spleen, bone marrow, kidney, liver, and other organs, resulting in hemosiderosis. In a healthy person, a red blood cell survives 90 to 120 days in the...
ISBN 978-0-12-383864-3. S2CID 90250940. Richter, Goetz (1 August 1957). "A Study of Hemosiderosis with Aid of Electron Microscopy". The Journal of Experimental Medicine...
spleen, bone marrow, kidney, liver, and other organs, resulting in hemosiderosis. In vitro hemolysis can be caused by improper technique during collection...
Superficial hemosiderosis of the central nervous system is a disease of the brain resulting from chronic iron deposition in neuronal tissues associated...
examination showed the cause of death was hemosiderosis due to hepatic iron accumulation from his many blood transfusions and iron supplements. James A. Halsted...
Retrieved 20 November 2021. MacDonald RA (1964). Hemochromatosis and Hemosiderosis. Springfield, IL: Charles C Thomas. Simon M, Bourel M, Fauchet R, Genetet...
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